neurodegeneration

It takes two: Genes ATP13A2 and GBA1 interact to drive neurodegeneration

by Homa Warren, Baylor College of Medicine edited by Gaby Clark, reviewed by Andrew Zinin  Editors’ notes  The GIST Add as preferred source Credit: CC0 Public Domain Parkinson’s disease (PD) is the second most common neurodegenerative disease after Alzheimer’s disease, affecting more than 10 million people worldwide. People with this condition may experience tremors, limb stiffness, gait and […]

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Could glial cells be the missing link in treating neurodegeneration?

A new research highlight reveals glial cells as a promising frontier for neurodegeneration therapy Peer-Reviewed Publication Chinese Medical Journals Publishing House Co., Ltd. image: The figure illustrates the potential of targeting glial cell functions and their interactions with neurons as a therapeutic strategy for neurodegeneration. view more   Credit: Tobiloba S. Olajide from University of Medical Sciences.

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BU study of young athletes finds neurodegeneration might begin before CTEa

These results have the potential to significantly change our perspective on contact sports. Peer-Reviewed Publication Boston University School of Medicine EMBARGOED by Nature until 11 a.m. ET, September 17, 2025 Contact: Gina DiGravio, 617-358-7838, ginad@bu.edu BU Study of Young Athletes Finds Neurodegeneration Might Begin Before CTE These results have the potential to significantly change our perspective

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Metabolic signals in neurons determine whether axons degrade or resist neurodegeneration, study finds

by University of Michigan edited by Sadie Harley, reviewed by Andrew Zinin Graphical abstract. Credit: Molecular Metabolism (2025). DOI: 10.1016/j.molmet.2025.102187 Unlike most cells in the human body, neurons—the functional cells of our nervous system—cannot typically replace themselves with healthy copies after being damaged. Rather, after an injury from something like a stroke, concussion or neurodegenerative disease, neurons and their axons,

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